Affinage

EIF3K

Eukaryotic translation initiation factor 3 subunit K · UniProt Q9UBQ5

Length
218 aa
Mass
25.1 kDa
Annotated
2026-06-09
71 papers in source corpus 10 papers cited in narrative 10 extracted findings
Cross-family judge vs UniProt: Affinage preferred faithfulness: 6/6 claims corpus-supported (100%)

Mechanistic narrative

Synthesis pass · prose summary of the discoveries below

EIF3K is the smallest subunit of the mammalian eIF3 translation initiation complex, integrating into the holocomplex through direct binary contacts with eIF3c, eIF3g, and eIF3j and co-sedimenting with eIF3 on 40S ribosomal subunits (PMID:14519125). Its crystal structure resolves two domains—a HEAT-analogous (HAM) domain and a winged-helix-like (WH) domain—that present discrete protein- and putative RNA-binding surfaces (PMID:15180986). Functionally, EIF3K is not a core processivity factor but an mRNA-selective regulator: together with eIF3L it forms a module that binds the 5'-UTR of RPS15A mRNA to repress its translation, so that acute EIF3K depletion de-represses RPS15A synthesis and thereby promotes global translation, proliferation, tumor growth, and stress resistance (PMID:37155573). Consistent with this restraining role, loss of the EIF3K ortholog in C. elegans extends lifespan via DAF-16/FOXO and confers ER-stress resistance independent of the canonical UPR branches and of DAF-16, without altering bulk protein synthesis (PMID:27690135). Beyond the ribosome, EIF3K engages partners across compartments—cyclin D3 (PMID:15327989), the 5-HT7 receptor (PMID:16935469), PML nuclear bodies via a dedicated isoform (PMID:24036361), and cytoplasmic dynein intermediate chain at actin-dependent cortical sites of cell-cell contact (PMID:12006665)—and acts as an autophagic adaptor bridging K27-ubiquitinated MyD88 to ATG5 to suppress NF-κB innate immune signaling (PMID:35176284). Homozygous loss-of-function EIF3K variants underlie a neurodevelopmental syndrome (PMID:40219605).

Mechanistic history

Synthesis pass · year-by-year structured walk · 10 steps
  1. 2002 Medium

    Before its assignment to eIF3, the protein (PLAC-24) was first characterized as a direct dynein-associated factor, establishing an extra-ribosomal cytoskeletal/junctional context for EIF3K.

    Evidence Protein interaction screen and immunocytochemistry with cytoskeletal disruption and beta-catenin overexpression in epithelial cells

    PMID:12006665

    Open questions at the time
    • Functional consequence of dynein binding for translation or transport unresolved
    • Relationship between cortical recruitment and eIF3 holocomplex membership unclear
  2. 2003 High

    Identifying EIF3K as the twelfth eIF3 subunit with defined binary contacts placed it structurally within the initiation machinery and established its core interaction partners.

    Evidence Co-IP, affinity purification, sucrose gradient cosedimentation, baculovirus reconstitution, and GST pull-down in mammalian/insect systems

    PMID:14519125

    Open questions at the time
    • Whether EIF3K is required for eIF3 activity not tested
    • Functional role distinct from scaffolding not addressed
  3. 2004 High

    The crystal structure defined EIF3K's two-domain architecture and mapped candidate binding surfaces, providing the structural basis for its protein and RNA interactions.

    Evidence X-ray crystallography with structural comparison and sequence conservation analysis

    PMID:15180986

    Open questions at the time
    • RNA-binding activity inferred from structure but not demonstrated biochemically
    • Specific partners contacting each surface not identified structurally
  4. 2004 Medium

    Linking EIF3K to cyclin D3 connected it to cell-cycle-coupled control of translational output.

    Evidence Yeast two-hybrid, in vitro binding, co-IP, and confocal colocalization in human cells

    PMID:15327989

    Open questions at the time
    • Whether cyclin D3 acts through EIF3K to upregulate translation not directly shown
    • Nuclear vs cytoplasmic function of the interaction unresolved
  5. 2006 Medium

    Demonstrating EIF3K binding to the 5-HT7 receptor and driving its plasma-membrane relocalization extended its extra-ribosomal roles to receptor expression and trafficking.

    Evidence Yeast two-hybrid, co-IP, deletion mapping, and immunofluorescence in COS-7 cells

    PMID:16935469

    Open questions at the time
    • Mechanism by which EIF3K increases receptor levels (translation vs stability) not distinguished
    • Physiological relevance in neurons not tested
  6. 2013 Medium

    Identifying an EIF3K isoform that targets PML nuclear bodies linked the protein to a nuclear tumor-suppressor compartment.

    Evidence Yeast two-hybrid with PML-I bait, in vitro pull-down, co-IP, and co-immunofluorescence in human cells

    PMID:24036361

    Open questions at the time
    • Function of EIF3K at PML bodies unknown
    • Whether isoform-specific localization affects translation unexamined
  7. 2016 High

    Genetic loss of the EIF3K ortholog extending lifespan and ER-stress resistance without altering bulk translation revealed a dedicated regulatory, rather than essential, role for the accessory subunit.

    Evidence Loss-of-function genetics, lifespan and stress assays, and epistasis with DAF-16/UPR mutants in C. elegans

    PMID:27690135

    Open questions at the time
    • Molecular target mediating lifespan/stress effects not identified in this study
    • DAF-16-dependent lifespan vs DAF-16-independent stress resistance not mechanistically reconciled
  8. 2022 Medium

    Defining EIF3K as an autophagic adaptor bridging K27-ubiquitinated MyD88 to ATG5 established a translation-independent role in terminating NF-κB innate immune signaling.

    Evidence Co-IP, ubiquitination assays, autophagy flux assays, and gain/loss-of-function in teleost fish

    PMID:35176284

    Open questions at the time
    • Conservation of the MyD88-EIF3K-ATG5 axis in mammals not established
    • Domain of EIF3K mediating ubiquitin/ATG5 bridging not mapped
  9. 2023 High

    Showing that the EIF3K/eIF3L module represses RPS15A translation via its 5'-UTR provided the central mechanism: EIF3K is an mRNA-selective brake on ribosome biogenesis whose loss drives anabolic, pro-growth output.

    Evidence Multiomic profiling, acute subunit depletion, ectopic RPS15A rescue, 5'-UTR binding disruption, and mathematical modeling

    PMID:37155573

    Open questions at the time
    • Direct RNA contact by EIF3K vs eIF3L not separated
    • Signal coupling stress to EIF3K/eIF3L downregulation undefined
  10. 2025 Medium

    Identifying homozygous loss-of-function EIF3K variants in a neurodevelopmental syndrome established the gene's physiological requirement in humans.

    Evidence Exome/genome sequencing, RT-PCR splicing analysis, Western blot of patient fibroblasts, and familial segregation

    PMID:40219605

    Open questions at the time
    • Cellular pathway linking EIF3K loss to neurodevelopmental phenotype not defined
    • Whether RPS15A de-repression contributes to disease unexplored

Open questions

Synthesis pass · forward-looking unresolved questions
  • How EIF3K's many extra-ribosomal interactions (dynein, cyclin D3, 5-HT7, PML, MyD88-ATG5) integrate with its core role as an mRNA-selective translational repressor remains unresolved.
  • No unified model linking ribosomal and non-ribosomal functions
  • Tissue- and isoform-specific deployment of EIF3K functions uncharacterized
  • Mammalian validation of the autophagy/NF-κB role absent

Mechanism profile

Synthesis pass · controlled-vocabulary classification · explore literature graph →
Molecular activity
GO:0003723 RNA binding 2 GO:0045182 translation regulator activity 2 GO:0060090 molecular adaptor activity 1 GO:0098772 molecular function regulator activity 1
Localization
GO:0005634 nucleus 2 GO:0005829 cytosol 2 GO:0005886 plasma membrane 2 GO:0005840 ribosome 1
Pathway
R-HSA-168256 Immune System 1 R-HSA-9612973 Autophagy 1
Partners
CCND3EIF3CEIF3GEIF3JEIF3LHTR7MYD88PML
Complex memberships
eIF3

Evidence

Reading pass · 10 per-paper findings extracted from the source corpus
Year Finding Method Journal Conf PMIDs
2004 Crystal structure of human eIF3K was solved, revealing two distinct domains: a HEAT analogous motif (HAM) domain and a winged-helix-like (WH) domain. Structural comparison and sequence conservation analysis identified three putative protein-binding surfaces and potential RNA binding activity. X-ray crystallography with structural comparison and sequence conservation analysis The Journal of biological chemistry High 15180986
2003 eIF3K (28 kDa) was identified as the twelfth subunit of mammalian eIF3 complex. It co-immunoprecipitates with the eIF3 complex, co-purifies with other eIF3 subunits upon affinity purification, colocalizes with eIF3 on 40S ribosomal subunits, and forms a stable complex with core eIF3 subunits when co-expressed in baculovirus-infected insect cells. Direct binary interactions were established with eIF3c, eIF3g, and eIF3j by GST pull-down assays. Co-immunoprecipitation, affinity purification, sucrose gradient sedimentation, baculovirus co-expression, GST pull-down assay European journal of biochemistry High 14519125
2004 eIF3K (p28 subunit) interacts with cyclin D3. The interaction was identified by yeast two-hybrid screen of a human fetal liver cDNA library, confirmed by in vitro binding assay, co-immunoprecipitation in vivo, and co-localization by confocal microscopy. Cyclin D3 interacts with eIF3K through its C-terminal domain. eIF3K localizes to both nucleus and cytoplasm and co-localizes with cyclin D3. Overexpression of cyclin D3 upregulates cellular translation activity without changing mRNA levels. Yeast two-hybrid, in vitro binding assay, co-immunoprecipitation, confocal immunofluorescence FEBS letters Medium 15327989
2006 eIF3K (PLAC-24) interacts with all three isoforms of the 5-HT7 receptor. Interaction was identified by yeast two-hybrid screen and confirmed by co-immunoprecipitation in transfected COS-7 cells. The interaction is not restricted to the receptor C-terminus. Both the HAM and WH domains of eIF3K interact with the 5-HT7(a) receptor. Overexpression of eIF3K causes a ~3-fold increase in 5-HT7(a) receptor expression levels. Co-expression with the receptor causes eIF3K to relocate from nucleus/perinuclear sites to the plasma membrane, suggesting a role in receptor transport and stabilization. Yeast two-hybrid, co-immunoprecipitation, deletion mutagenesis, immunofluorescence co-localization Cellular signalling Medium 16935469
2002 PLAC-24 (eIF3K) binds directly to cytoplasmic dynein intermediate chain (DIC) and is not a dynactin subunit; the binding is independent of dynein-dynactin association. eIF3K shows a punctate perinuclear distribution in isolated cells but is specifically recruited to cortical sites of cell-cell contact in epithelial cells, where it co-localizes with adherens junction components. Cortical localization requires intact actin filaments but not microtubules (shown by latrunculin vs. nocodazole treatment). Overexpression of beta-catenin abolishes eIF3K localization to cell-cell contacts. Protein interaction screen, immunocytochemistry, cytoskeletal disruption assays (latrunculin, nocodazole), beta-catenin overexpression Molecular biology of the cell Medium 12006665
2013 eIF3K interacts with the PML tumor suppressor protein. Interaction was identified by yeast two-hybrid screen using PML isoform I (PML-I) peptide sequences as bait, and confirmed by in vitro pull-down and in vivo co-immunoprecipitation and co-immunofluorescence in human cells. A novel eIF3K isoform (eIF3K-2) was identified that specifically co-localizes to PML nuclear bodies, particularly those enriched in PML-I, while eIF3K isoform 1 associates poorly with PML NBs. This identifies eIF3K as the first known eIF3 subunit to interact directly with PML. Yeast two-hybrid, in vitro pull-down, co-immunoprecipitation, co-immunofluorescence Experimental cell research Medium 24036361
2016 Loss-of-function mutations in C. elegans eif-3.K (ortholog of EIF3K) extend lifespan by ~40% and confer enhanced resistance to ER stress without affecting bulk protein synthesis or growth rates. Lifespan extension requires the DAF-16 (FOXO) transcription factor. Enhanced ER stress resistance is independent of IRE-1-XBP-1, ATF-6, and PEK-1 pathways and also independent of DAF-16, indicating eIF3K and eIF3L accessory subunits have a distinct regulatory role in ER stress and aging responses. Loss-of-function genetics, lifespan assays, epistasis analysis with DAF-16, IRE-1, XBP-1, ATF-6, PEK-1 mutants, protein synthesis measurements PLoS genetics High 27690135
2022 In teleost fish, eIF3K acts as a suppressor of the NF-κB pathway. Mechanistically, eIF3K expression (induced by Vibrio harveyi) enhances E3 ligase Nrdp1-mediated K27-linked ubiquitination of MyD88. eIF3K then bridges ubiquitin-tagged MyD88 to ATG5, forming a MyD88-eIF3k-ATG5 complex that is transported to the autophagosome for degradation, thereby terminating innate immune signaling. Co-immunoprecipitation, ubiquitination assays, autophagy flux assays, overexpression and knockdown experiments The Journal of biological chemistry Medium 35176284
2023 Acute depletion of eIF3K promotes global translation, cell proliferation, tumor growth, and stress resistance by de-repressing the synthesis of ribosomal protein RPS15A. eIF3K and eIF3L form a mRNA-specific module that controls RPS15A translation by binding to the 5'-UTR of RPS15A mRNA; disruption of eIF3 binding to the 5'-UTR of RPS15A mRNA negated the anabolic effects of eIF3K depletion. Ectopic expression of RPS15A mimicked the anabolic effects of eIF3K depletion. eIF3K and eIF3L are selectively downregulated in response to ER and oxidative stress, suggesting this module acts as a rheostat of ribosome content. Multiomic profiling (proteomics, translatome), acute depletion of eIF3 subunits, ectopic RPS15A expression, 5'-UTR binding disruption experiments, mathematical modeling The EMBO journal High 37155573
2025 Homozygous variants in EIF3K (missense p.Asp43Gly and intronic c.355-13A>G) are associated with a neurodevelopmental syndrome. The intronic variant causes aberrant splicing of EIF3K pre-mRNA (insertion of 12 intronic base pairs, skipping of 2 exons) and significantly reduced EIF3K protein levels in patient skin fibroblasts, establishing that loss of EIF3K function underlies the phenotype. Whole exome/genome sequencing, RT-PCR splicing analysis, Western blot of patient fibroblasts, familial segregation analysis HGG advances Medium 40219605

Source papers

Stage 0 corpus · 71 papers · ranked by NIH iCite citations
Year Title Journal Citations PMID
1997 A role for the M9 transport signal of hnRNP A1 in mRNA nuclear export. The Journal of cell biology 222 9105034
1999 Nup153 is an M9-containing mobile nucleoporin with a novel Ran-binding domain. The EMBO journal 198 10202161
2021 Adjunctive Probiotic Lactobacillus rhamnosus Probio-M9 Administration Enhances the Effect of Anti-PD-1 Antitumor Therapy via Restoring Antibiotic-Disrupted Gut Microbiota. Frontiers in immunology 87 34970262
1992 M4 and M9 antibodies in the overlap syndrome of primary biliary cirrhosis and chronic active hepatitis: epitopes or epiphenomena? Hepatology (Baltimore, Md.) 61 1330864
2004 Crystal structure of human eIF3k, the first structure of eIF3 subunits. The Journal of biological chemistry 55 15180986
2008 Efficient purification of the biosurfactant viscosin from Pseudomonas libanensis strain M9-3 and its physicochemical and biological properties. Journal of natural products 51 18471020
2003 Characterization of eIF3k: a newly discovered subunit of mammalian translation initiation factor elF3. European journal of biochemistry 51 14519125
2016 Mutations in Nonessential eIF3k and eIF3l Genes Confer Lifespan Extension and Enhanced Resistance to ER Stress in Caenorhabditis elegans. PLoS genetics 44 27690135
1999 A novel insect V-ATPase subunit M9.7 is glycosylated extensively. The Journal of biological chemistry 39 10358099
2022 Lacticaseibacillus rhamnosus Probio-M9 extends the lifespan of Caenorhabditis elegans. Communications biology 38 36302976
2006 Two motifs essential for nuclear import of the hnRNP A1 nucleocytoplasmic shuttling sequence M9 core. FEBS letters 37 16455081
1985 The pathogenicity of the A7, M9 and L10 strains of Semliki Forest virus for weanling mice and primary mouse brain cell cultures. The Journal of general virology 35 2997371
1984 The pathogenicity of the M9 mutant of Semliki Forest virus in immune-compromised mice. The Journal of general virology 32 6319575
1983 Oligodendrocyte infection and demyelination produced in mice by the M9 mutant of Semliki Forest virus. Acta neuropathologica 30 6310927
2022 eIF3k inhibits NF-κB signaling by targeting MyD88 for ATG5-mediated autophagic degradation in teleost fish. The Journal of biological chemistry 28 35176284
1990 Anti-M9 antibodies in sera from patients with primary biliary cirrhosis recognize an epitope of glycogen phosphorylase. Clinical and experimental immunology 27 1696184
1983 Description of a new Qa antigenic specificity, "Qa-m9," whose expression is under complex genetic control. Journal of immunology (Baltimore, Md. : 1950) 27 6193187
2005 Thr-774 (transmembrane segment M5), Val-920 (M8), and Glu-954 (M9) are involved in Na+ transport, and Gln-923 (M8) is essential for Na,K-ATPase activity. The Journal of biological chemistry 26 15764602
2002 Single nucleotide polymorphism genotyping by on-line liquid chromatography-mass spectrometry in forensic science of the Y-chromosomal locus M9. Journal of chromatography. B, Analytical technologies in the biomedical and life sciences 26 12457998
2021 Lactobacillus rhamnosus Probio-M9 Improves the Quality of Life in Stressed Adults by Gut Microbiota. Foods (Basel, Switzerland) 23 34681433
2017 The molecular mechanisms of Monascus purpureus M9 responses to blue light based on the transcriptome analysis. Scientific reports 23 28717254
2016 Obtaining Soluble Folded Proteins from Inclusion Bodies Using Sarkosyl, Triton X-100, and CHAPS: Application to LB and M9 Minimal Media. Current protocols in protein science 23 27038270
2012 Kinetic expression analysis of the cluster mdv1-mir-M9-M4, genes meq and vIL-8 differs between the lytic and latent phases of Marek's disease virus infection. The Journal of general virology 22 22442112
2004 Identification of the p28 subunit of eukaryotic initiation factor 3(eIF3k) as a new interaction partner of cyclin D3. FEBS letters 21 15327989
2023 eIF3 mRNA selectivity profiling reveals eIF3k as a cancer-relevant regulator of ribosome content. The EMBO journal 20 37155573
2002 PLAC-24 is a cytoplasmic dynein-binding protein that is recruited to sites of cell-cell contact. Molecular biology of the cell 19 12006665
2023 Lactobacillus rhamnosus Probio-M9 alleviates OVA-sensitized food allergy through modulating gut microbiota and its metabolism. Food & function 18 37982421
2021 eIF3k Domain-Containing Protein Regulates Conidiogenesis, Appressorium Turgor, Virulence, Stress Tolerance, and Physiological and Pathogenic Development of Magnaporthe oryzae Oryzae. Frontiers in plant science 18 34733303
1997 Sinus-lining cells of the lymph nodes recognized as a dendritic cell type by the new monoclonal antibody Ki-M9. The American journal of pathology 17 9250155
2018 Enhanced Metabolite Productivity of Escherichia coli Adapted to Glucose M9 Minimal Medium. Frontiers in bioengineering and biotechnology 16 30483499
2015 A novel eurythermic and thermostale lipase LipM from Pseudomonas moraviensis M9 and its application in the partial hydrolysis of algal oil. BMC biotechnology 16 26463643
2006 Novel interaction between the human 5-HT7 receptor isoforms and PLAC-24/eIF3k. Cellular signalling 16 16935469
2020 Development of a M9-based urea medium (M9U) for sensitive and real-time monitoring of ureolytic activity of bacteria and cell-free urease. MicrobiologyOpen 15 31943918
2020 Neisseria gonorrhoeae-induced salpingitis is targeted by circular RNA EIF3K via miR-139-5p and regulating MAPK/NF-κB signaling pathway to promotes apoptosis and autophagy bacterial cells. Microbial pathogenesis 14 32045642
2019 The Effect of Blue Light on the Production of Citrinin in Monascus purpureus M9 by Regulating the mraox Gene through lncRNA AOANCR. Toxins 13 31540336
2013 The translation initiation factor 3 subunit eIF3K interacts with PML and associates with PML nuclear bodies. Experimental cell research 13 24036361
2013 M9, a novel region of amino-Nogo-A, attenuates cerebral ischemic injury by inhibiting NADPH oxidase-derived superoxide production in mice. CNS neuroscience & therapeutics 12 23490284
2003 A novel putative M9.2 isoform of V-ATPase expressed in the nervous system. Neuroreport 12 12544825
2025 Nano-Zinc Oxide Can Enhance the Tolerance of Apple Rootstock M9-T337 Seedlings to Saline Alkali Stress by Initiating a Variety of Physiological and Biochemical Pathways. Plants (Basel, Switzerland) 11 39861585
2021 AhaP, A Quorum Quenching Acylase from Psychrobacter sp. M9-54-1 That Attenuates Pseudomonas aeruginosa and Vibrio coralliilyticus Virulence. Marine drugs 11 33401388
2023 The Space Environment Activates Capsular Polysaccharide Production in Lacticaseibacillus rhamnosus Probio-M9 by Mutating the wze (ywqD) Gene. Microbiology spectrum 10 36861974
2023 Trace impurities in sodium phosphate influences the physiological activity of Escherichia coli in M9 minimal medium. Scientific reports 10 37833342
2021 Ammonium nitrate regulated the color characteristic changes of pigments in Monascus purpureus M9. AMB Express 10 33398480
2018 MdPIN1b encodes a putative auxin efflux carrier and has different expression patterns in BC and M9 apple rootstocks. Plant molecular biology 10 29340953
2014 Loop-mediated isothermal amplification assay targeting the blaCTX-M9 gene for detection of extended spectrum β-lactamase-producing Escherichia coli and Klebsiella pneumoniae. Microbiology and immunology 10 25284314
2004 Matrix metalloproteinase 9 expression is coordinately modulated by the KRE-M9 and 12-O-tetradecanoyl-phorbol-13-acetate responsive elements. The Journal of investigative dermatology 10 15009705
1998 A protein (M9) associated with monoclonal antibody-mediated agglutination of Mycoplasma gallisepticum is a member of the pMGA family. Infection and immunity 10 9784576
1985 Variable expression of Qa-m7, Qa-m8, and Qa-m9 antigenic determinants on primitive hemopoietic precursor cells. Journal of cellular physiology 10 2580849
2024 X-ray structure and characterization of a probiotic Lactobacillus rhamnosus Probio-M9 L-rhamnose isomerase. Applied microbiology and biotechnology 8 38430263
2014 Extracellular proteases of Halobacillus blutaparonensis strain M9, a new moderately halophilic bacterium. Brazilian journal of microbiology : [publication of the Brazilian Society for Microbiology] 8 24688526
2025 Highly Efficient Biosynthesis of Rebaudioside M9 through Enzyme Screening and Structure-Guided Engineering. Journal of agricultural and food chemistry 6 39882950
2024 Disruption of UDP-galactopyranose mutase expression: A novel strategy for regulation of galactomannan biosynthesis and monascus pigments secretion in Monascus purpureus M9. International journal of biological macromolecules 6 38218271
2024 Long-Term Tracking of the Effects of Colostrum-Derived Lacticaseibacillus rhamnosus Probio-M9 on Gut Microbiota in Mice with Colitis-Associated Tumorigenesis. Biomedicines 6 38540144
2024 Carbon metabolism of a novel isolate from Lacticaseibacillus rhamnosus Probio-M9 derived through space mutant. Journal of applied microbiology 5 39152088
2002 A nuclear targeting peptide, M9, improves transfection efficiency in fibroblasts. The Journal of surgical research 5 12443719
2014 Induction of viable but non-culturable (VBNC) state in Salmonella cultured in M9 minimal medium containing high glucose. Biological & pharmaceutical bulletin 4 25109306
2008 Physiological dissection of blue and red light regulation of apical dominance and branching in M9 apple rootstock growing in vitro. Journal of plant physiology 4 18423933
2024 Effects of combined application of phosphorus and zinc on growth and physiological characteristics of apple rootstock M9-T337 seedlings (Malus domestica Borkh.). BMC plant biology 3 39448942
1990 Antigenicity of group B Streptococcus strain "M9" and its prevalence in Japan. Kansenshogaku zasshi. The Journal of the Japanese Association for Infectious Diseases 3 2193068
2025 A Sensitive and Selective LC-MS/MS-ESI Method for the Quantitation of Metabolites M9, M12, and M20 of Bexicaserin in Human Plasma and Urine Matrices. Biomedical chromatography : BMC 2 39968937
2025 Engineering saline-alkali-tolerant apple rootstocks by overexpressing MdHYL1 in M9-T337. Molecular breeding : new strategies in plant improvement 2 40583866
2020 Nonmetabolizable Arabinose Inhibits Vibrio cholerae Growth in M9 Medium with Gluconate as the Sole Carbon Source. Japanese journal of infectious diseases 2 32350213
2002 Cloning of a complementary DNA encoding the unique dendritic cell antigen Ki-M9. Cell and tissue research 2 11904771
2025 Homozygous variants in EIF3K associated with neurodevelopmental delay, microcephaly, and growth retardation. HGG advances 1 40219605
2025 Structural and Immunomodulatory Changes in Mycelial and Exopolysaccharides from Monascus purpureus M9 after Mutation of the α-1,3-Glucan Synthase Gene. Journal of agricultural and food chemistry 1 40322926
2025 Engineering saline-alkali-tolerant apple rootstock by knocking down MdGH3 genes in M9-T337. Stress biology 1 40549263
2025 Lacticaseibacillus rhamnosus Probio-M9 Alters the Gut Microbiota and Mitigates Pulmonary Hypertension in a Rat Model. Nutrients 1 41010453
2021 Corrigendum: eIF3k Domain-Containing Protein Regulates Conidiogenesis, Appressorium Turgor, Virulence, Stress Tolerance, and Physiological and Pathogenic Development of Magnaporthe oryzae Oryzae. Frontiers in plant science 1 34899817
2026 M9 medium composition alters E. coli metabolism during recombinant expression. Journal of biomolecular NMR 0 41701392
2025 Recombinant α-Toxin BmK-M9 Inhibits Breast Cancer Progression by Regulating β-Catenin In Vivo. Cell biochemistry and biophysics 0 40080350
1995 [Genetic and molecular analysis of new recombinant t-haplotypes t(M8) and t(M9)]. Genetika 0 7607427

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