Affinage

RAB11FIP1

Rab11 family-interacting protein 1 · UniProt Q6WKZ4

Length
1283 aa
Mass
137.2 kDa
Annotated
2026-06-10
34 papers in source corpus 17 papers cited in narrative 18 extracted findings
Cross-family judge vs UniProt: Affinage preferred faithfulness: 5/5 claims corpus-supported (100%)

Mechanistic narrative

Synthesis pass · prose summary of the discoveries below

RAB11FIP1 (Rab11-FIP1/RCP) is a Rab11a effector that organizes cargo recycling through the slow endocytic recycling compartment (PMID:11495908). It binds active Rab11a, as well as Rab11b and Rab25, via a conserved C-terminal amphipathic alpha-helix that is required for the interaction (PMID:11495908), and is expressed as multiple alternatively spliced isoforms (FIP1A-H) of which only those retaining this domain target Rab11a-positive recycling membranes, occupying spatially and temporally distinct tubular subdomains and forming selective pairwise associations with one another (PMID:16920206, PMID:23283983). Functionally, RAB11FIP1 channels cargo from early endosomes (Rab5/EEA1-positive) into the Rab11a recycling system, and a dominant-negative T197A mutation collapses recycling endosomes, sequesters Rab11a and transferrin receptor, and blocks transferrin recycling (PMID:26790954); in vivo it is required for proper recycling of the transmembrane mucin MUC13, whose loss in knockout mice causes MUC13/Rab14 mistrafficking and spontaneous colonic inflammation (PMID:35819177). RAB11FIP1 activity is set by post-translational control: MARK2 phosphorylates it at S234 to govern epithelial polarity (PMID:28396819), the E3 ligase RFFL directly ubiquitylates it (PMID:30659120), TRIM21 adds K11-linked polyubiquitin to promote pIgR/pIgA transcytosis (PMID:34638806), and its proteasomal turnover can be blocked by circRTN4 (PMID:34983537). In cancer, RAB11FIP1 acts as an oncogenic transformer that activates Ras/ERK signaling and drives invasion and metastasis (PMID:19620787), while in epithelial contexts it suppresses EMT by inhibiting Zeb1 (PMID:33403789); through its control of transferrin receptor recycling it governs iron uptake and ferroptosis sensitivity, a function antagonized by RAB11FIP5-mediated sequestration of Rab11a (PMID:42207639).

Mechanistic history

Synthesis pass · year-by-year structured walk · 13 steps
  1. 2001 High

    Established RAB11FIP1 as a bona fide Rab11a effector and defined the structural basis of the interaction, anchoring it to the membrane recycling machinery.

    Evidence Yeast two-hybrid, Co-IP, colocalization, and parietal cell fractionation; domain deletion mapping the C-terminal amphipathic alpha-helix

    PMID:11495908

    Open questions at the time
    • Did not define which cargoes traffic through FIP1-Rab11a domains
    • Functional consequence of binding for recycling kinetics not addressed
  2. 2003 Medium

    Showed RAB11FIP1 localization is cell-cycle regulated, distinguishing it from other Rab11 effectors by its mitotic relocation to pericentriolar material.

    Evidence Live and fixed fluorescence microscopy of GFP-RAB11FIP1 across cell cycle stages with membrane fractionation in MDCK cells

    PMID:12956871

    Open questions at the time
    • Mechanism driving mitotic relocation unknown
    • Functional role of pericentriolar targeting not established
  3. 2006 Medium

    Resolved that only Rab11-binding-domain-containing isoforms target recycling membranes, and that endogenous isoforms occupy distinct subcompartments.

    Evidence Cloning of eight splice variants, GFP-fusion colocalization, isoform-specific antibody staining in HeLa

    PMID:16920206

    Open questions at the time
    • Distinct functions of individual isoforms not defined
    • Regulation of splicing not addressed
  4. 2009 High

    Identified RAB11FIP1/RCP as an oncogenic driver coupling vesicle recycling to Ras/ERK signaling, transformation, and metastasis.

    Evidence Overexpression and shRNA in mammary/breast cancer lines, xenografts, ERK phosphorylation and Ras activation assays

    PMID:19620787

    Open questions at the time
    • Molecular link between recycling activity and Ras activation not resolved
    • Relevant trafficked cargo driving transformation not identified
  5. 2013 Medium

    Defined the spatiotemporal organization of FIP1 isoforms within recycling tubules and their hetero-associations, plus a cargo-specific role in adiponectin secretion.

    Evidence Live-cell deconvolution imaging with transferrin pulse-chase; overexpression/knockdown with adiponectin ELISA in HEK293 and 3T3-L1

    PMID:23283983 PMID:24040321

    Open questions at the time
    • How cargo selectivity (adiponectin vs transferrin) is achieved unclear
    • Basis for distinct subdomain occupancy not mechanistically defined
  6. 2014 Medium

    Linked FIP1-dependent recycling to phosphatidylserine distribution, implicating lipid handling in compartment organization.

    Evidence Live-cell and structured illumination microscopy with LactC2-PS biosensor and C2-domain deletion

    PMID:25210648

    Open questions at the time
    • Whether FIP1 directly binds PS not established
    • Functional consequence of PS redistribution unknown
  7. 2016 Medium

    Demonstrated that FIP1A controls transit from early endosomes into the Rab11a recycling system, with a dominant-negative mutant blocking transferrin recycling.

    Evidence T197A site-directed mutagenesis, GFP-fusion imaging, transferrin recycling assay, marker colocalization (Rab5, EEA1)

    PMID:26790954

    Open questions at the time
    • Molecular role of T197 not defined (not a known phospho-site here)
    • Direct early-endosome-to-FIP1 handoff machinery unknown
  8. 2017 Medium

    Identified MARK2-mediated S234 phosphorylation as a switch controlling FIP1 function in epithelial apical-basal polarity.

    Evidence Kinase assay, phospho-specific antibody, calcium-switch repolarization, phospho-mutant overexpression with polarity marker imaging in MDCK

    PMID:28396819

    Open questions at the time
    • How S234 phosphorylation alters Rab11a binding or cargo handling not resolved
    • Upstream signals activating MARK2 in this context unknown
  9. 2019 High

    Established RFFL as a direct E3 ligase for FIP1, placing FIP1 activity under ubiquitin control during recycling.

    Evidence In vitro ubiquitylation reconstitution, RFFL knockout cells, BioID interactome, dominant-negative RFFL with transferrin recycling assay

    PMID:30659120

    Open questions at the time
    • Ubiquitin linkage type and degradative vs non-degradative outcome not defined
    • Which FIP1 lysines are modified unknown
  10. 2021 Medium

    Revealed context-dependent roles: FIP1 suppresses EMT via Zeb1 inhibition in ESCC, and is activated by TRIM21 K11-polyubiquitination to drive pIgR/pIgA transcytosis.

    Evidence shRNA in 3D organoids and ESCC lines with EMT markers and mouse model; siRNA, Co-IP, linkage-specific ubiquitin antibodies and transcytosis assays

    PMID:33403789 PMID:34638806

    Open questions at the time
    • Mechanism by which FIP1 inhibits Zeb1 not defined
    • Reconciliation of tumor-suppressive (ESCC) vs oncogenic (breast) roles unresolved
  11. 2022 High

    Defined an in vivo physiological role in colonic mucin recycling and showed circRNA-mediated stabilization of FIP1 against proteasomal degradation.

    Evidence Global KO mouse with immunostaining/RNA-seq/DSS colitis; circRTN4 pulldown-MS, protein stability and ubiquitination assays in pancreatic cancer cells

    PMID:34983537 PMID:35819177

    Open questions at the time
    • How FIP1 directs MUC13 away from Rab14 not mechanistically defined
    • circRTN4 binding site on FIP1 vs ubiquitination site not precisely mapped
  12. 2023 Medium

    Extended FIP1 functions to extracellular vesicle recycling under hypoxia and to transcriptional induction during Schwann cell differentiation.

    Evidence siRNA knockdown with Rab11a-sEV Co-IP and rat IVDD model; Stat1 ChIP-seq, luciferase reporter and knockdown in Schwann cells with sciatic nerve model

    PMID:36730125 PMID:37365519

    Open questions at the time
    • Direct role of FIP1 in sEV cargo selectivity unclear
    • Downstream FIP1 effectors in myelination not identified
  13. 2026 Medium

    Showed that FIP1-mediated transferrin receptor recycling governs iron uptake and ferroptosis, with RAB11FIP5 antagonizing FIP1 by sequestering Rab11a.

    Evidence RAB11FIP5 knockout cells, transferrin recycling and iron uptake assays, ferroptosis assays, xenografts in HNSCC

    PMID:42207639

    Open questions at the time
    • Direct competition for Rab11a not structurally resolved
    • Whether other FIPs similarly modulate ferroptosis untested

Open questions

Synthesis pass · forward-looking unresolved questions
  • It remains unresolved how RAB11FIP1's many post-translational modifications and isoform/competition states are integrated to switch between oncogenic, tumor-suppressive, and homeostatic recycling outputs in different tissues.
  • No unified model linking S234 phosphorylation, RFFL/TRIM21 ubiquitination, and FIP-FIP competition
  • Structural basis for cargo selectivity unknown
  • Reconciliation of opposing cancer phenotypes across tissues unaddressed

Mechanism profile

Synthesis pass · controlled-vocabulary classification · explore literature graph →
Molecular activity
GO:0008092 cytoskeletal protein binding 2 GO:0060090 molecular adaptor activity 2
Localization
GO:0031410 cytoplasmic vesicle 4 GO:0005768 endosome 1 GO:0005794 Golgi apparatus 1 GO:0005815 microtubule organizing center 1
Pathway
R-HSA-5653656 Vesicle-mediated transport 3 R-HSA-9609507 Protein localization 3 R-HSA-162582 Signal Transduction 1
Partners
MARK2RAB11ARAB11FIP3RAB11FIP5RAB25RFFLTRIM21

Evidence

Reading pass · 18 per-paper findings extracted from the source corpus
Year Finding Method Journal Conf PMIDs
2001 RAB11-FIP1 (Rab11-FIP1) was identified as a Rab11a-binding protein that associates with wild-type and dominant-active Rab11a (S20V), as well as Rab11b and Rab25, through a conserved carboxyl-terminal amphipathic alpha-helix. RAB11-FIP1 colocalizes with Rab11a in plasma membrane recycling systems in non-polarized HeLa cells and polarized MDCK cells, and co-enriches with Rab11a and H+K+-ATPase on parietal cell tubulovesicles, translocating with them upon histamine stimulation. Co-immunoprecipitation, yeast two-hybrid, colocalization by fluorescence microscopy, subcellular fractionation of parietal cells The Journal of biological chemistry High 11495908
2001 Binding of RAB11-FIP1 to Rab11a requires the conserved carboxyl-terminal amphipathic alpha-helix of RAB11-FIP1; this domain is necessary for Rab11 interaction. Domain deletion/mutation analysis combined with co-immunoprecipitation The Journal of biological chemistry High 11495908
2003 During mitosis in MDCK cells, RAB11-FIP1 dissociates from recycling system markers during late prophase and relocates to the pericentriolar material, then returns to recycling system vesicles during telophase, indicating a cell-cycle-regulated change in subcellular localization distinct from other Rab11 effectors. Live-cell and fixed fluorescence microscopy of GFP-tagged RAB11-FIP1 through cell cycle stages, western blot analysis of membrane association Traffic (Copenhagen, Denmark) Medium 12956871
2006 The RAB11FIP1 gene encodes eight alternatively spliced transcripts (FIP1A-H); isoforms A-D contain the C-terminal Rab11-binding domain and target to Rab11a-containing recycling membranes when overexpressed in HeLa cells, while isoforms E, F, and H lack the Rab11-binding domain and do not localize to recycling system membranes. Endogenous FIP1C/RCP occupies a partially distinct subcompartment within the recycling system compared to FIP1A and FIP1B. Cloning of alternatively spliced transcripts, GFP-fusion overexpression with colocalization microscopy, isoform-specific antibody staining Biochimica et biophysica acta Medium 16920206
2009 Overexpression of RCP/RAB11FIP1 in MCF10A normal mammary epithelial cells induces loss of contact inhibition, growth-factor independence, and anchorage-independent growth. Knockdown of RCP in breast cancer cell lines inhibits colony formation, invasion, and migration in vitro and reduces tumor formation and metastasis in mouse xenograft models. Overexpression of RCP enhances ERK phosphorylation and increases Ras activation in vitro. Overexpression and shRNA knockdown in cell lines, mouse xenograft tumor models, ERK phosphorylation assay, Ras activation assay The Journal of clinical investigation High 19620787
2013 RAB11-FIP1 isoforms (FIP1A, FIP1B, FIP1C) occupy spatially and temporally distinct subdomains within the Rab11a-dependent recycling system; FIP1B reaches maximal transferrin colocalization early (5 min), while FIP1A and FIP1C peak later (≥10 min). FIP1A directly associates with FIP1B and FIP1C, and also associates with FIP3-containing membranes. Rab11-FIP1 proteins form selective pairwise associations on dynamic tubular recycling compartments. Live-cell deconvolution fluorescence microscopy of GFP-tagged FIPs, transferrin pulse-chase assay, dual-expression live-cell imaging Molecular biology of the cell Medium 23283983
2013 RAB11-FIP1 (FIP1) overexpression decreases adiponectin secretion in HEK293 cells, and shRNA-mediated depletion of FIP1 enhances adiponectin release from 3T3-L1 adipocytes. Endogenous FIP1 co-distributes intracellularly with endogenous adiponectin. FIP1 depletion does not alter transferrin receptor recycling or insulin-mediated Glut4 trafficking. Overexpression and shRNA knockdown, adiponectin secretion ELISA, immunofluorescence colocalization, transferrin recycling assay PloS one Medium 24040321
2014 RAB11-FIP1 proteins (FIP1A, FIP1B, FIP1C) consistently colocalize with the phosphatidylserine (PS) probe LactC2 along peripheral and pericentriolar compartments. Removal of C2 domains from Rab11-FIPs causes accumulation of LactC2 probe in the pericentriolar region, suggesting that inhibition of recycling affects PS distribution. Live-cell fluorescence microscopy and structured illumination microscopy with LactC2-PS biosensor co-expressed with GFP-Rab11-FIPs Cellular logistics Medium 25210648
2016 A point mutation at Threonine-197 to Alanine in Rab11-FIP1A acts as a dominant-negative that collapses the normally distributed recycling endosomes into a central membranous cisternum, sequesters endogenous Rab11a, Rab11-FIP1C, and transferrin receptor (CD71), and causes a strong block in transferrin recycling. Rab5 and EEA1 (but not Rab4) accumulate in FIP1A-positive membranes, suggesting a direct pathway from early endosomes into the Rab11a recycling system. Site-directed mutagenesis, GFP-fusion overexpression, fluorescence microscopy, transferrin recycling assay, nocodazole treatment Experimental cell research Medium 26790954
2017 MARK2 kinase phosphorylates Rab11-FIP1B/C at serine 234 (in a consensus site analogous to the previously identified S227 in Rab11-FIP2). In MDCK cells during calcium-switch-induced repolarization, pS234-FIP1 phosphorylation persists through and after polarity reestablishment. Overexpression of non-phosphorylatable FIP1C(S234A) induces a lateral lumen phenotype with misorientation of apical markers (ezrin, syntaxin 3, podocalyxin), F-actin, ZO1, centrosome, and Golgi apparatus, demonstrating that FIP1 phosphorylation by MARK2 regulates epithelial polarity. Phospho-specific antibody generation, kinase assay, calcium-switch repolarization model, overexpression of phospho-mutants with immunofluorescence microscopy Cellular logistics Medium 28396819
2019 The ubiquitin ligase RFFL directly ubiquitylates Rab11-FIP1 in vitro, and RFFL knockout specifically reduces ubiquitylation of Rab11-FIP1 (while having minimal effect on ubiquitylation of EHD1, MICALL1, and Rab11-FIP2). RFFL interacts with class I Rab11-FIPs as identified by BioID proximity labeling. A dominant-negative RFFL mutant prolongs interaction with Rab11 effectors including FIP1, induces clustering of endocytic recycling compartments, and delays cargo recycling. BioID proximity proteomics, in vitro ubiquitylation assay, RFFL knockout cells, dominant-negative RFFL overexpression, transferrin recycling assay Journal of cell science High 30659120
2021 Genetic knockdown of Rab11-FIP1 in esophageal squamous cell carcinoma (ESCC) 3D organoid models increases organoid size and tumor cell invasion. Loss of Rab11-FIP1 in human ESCC cell lines decreases E-cadherin expression and increases mesenchymal markers (EMT). Rab11-FIP1 directly inhibits Zeb1, a key EMT transcription factor, thereby regulating EMT. shRNA knockdown, 3D organoid models, invasion assay, western blot for EMT markers, mouse model of ESCC EMBO reports Medium 33403789
2021 Rab11-FIP1 and Rab11-FIP5 knockdown additively impairs pIgR/pIgA transcytosis. TRIM21 mediates K11-linked polyubiquitination of Rab11-FIP1 (and K6-linked polyubiquitination of Rab11-FIP5) to promote their activation and facilitate pIgA transcytosis. In incompletely polarized cells, Rab11a-positive endosomes containing pIgR/pIgA, Rab11-FIP1, and Rab11-FIP5 are transported to the apical membrane via the Golgi apparatus. siRNA knockdown, co-immunoprecipitation, ubiquitin linkage-specific antibody analysis, transcytosis assay in polarized and incompletely polarized epithelial cells International journal of molecular sciences Medium 34638806
2022 Global Rab11FIP1 knockout (KO) mice develop spontaneous colonic inflammation with infiltration of macrophages and neutrophils, reduced TFF3-positive goblet cells, and abnormal accumulation of subapical vesicles in colonocytes. Rab11FIP1 loss causes mistrafficking of transmembrane mucin MUC13, which aberrantly internalizes with Rab14 instead of being properly recycled, resulting in impaired colonic mucosal integrity. Global KO mouse generation (exon 2 deletion), immunostaining, RNA-sequencing, DSS colitis model, confocal microscopy American journal of physiology. Gastrointestinal and liver physiology High 35819177
2022 CircRTN4 interacts with RAB11FIP1 protein to block its ubiquitination site, thereby stabilizing RAB11FIP1 and preventing its proteasomal degradation. CircRTN4 knockdown promotes RAB11FIP1 degradation by increasing its ubiquitination and decreases EMT marker expression (Slug, Snai1, Twist, Zeb1, N-cadherin) in pancreatic cancer cells. circRNA pulldown with mass spectrometry, protein stability assay, ubiquitination assay, 3D structure modeling Molecular cancer Medium 34983537
2023 HIF-1-induced upregulation of RCP/RAB11FIP1 under hypoxia promotes Rab11a-dependent recycling of internalized MSC-derived small extracellular vesicles (MSC-sEVs), reducing their intracellular cargo delivery efficiency. RAB11FIP1 enhances the interaction between Rab11a and MSC-sEVs under hypoxic conditions, and siRNA-mediated knockdown of RCP in engineered MSC-sEVs inhibits this recycling, improving cargo delivery. siRNA knockdown, co-immunoprecipitation (Rab11a-sEV interaction), rat intervertebral disk degeneration model, fluorescence microscopy of sEV trafficking ACS nano Medium 36730125
2023 The transcription factor Stat1 binds to the promoter of Rab11fip1 to initiate Schwann cell differentiation. Stat1 knockdown reduces Rab11fip1 expression and blocks Schwann cell differentiation, thereby inhibiting myelination. ChIP-seq, ChIP-qPCR, luciferase reporter assay, siRNA knockdown in Schwann cells, in vivo sciatic nerve model Molecular medicine (Cambridge, Mass.) Medium 37365519
2026 RAB11FIP5 competitively sequesters RAB11A to antagonize RAB11FIP1-mediated transferrin and transferrin receptor recycling in HNSCC cells, thereby restricting iron uptake and suppressing ferroptosis. Loss of RAB11FIP5 thus permits RAB11FIP1-mediated recycling, increasing iron uptake and ferroptosis sensitivity. RAB11FIP5 knockout cell lines, transferrin recycling assay, iron uptake measurement, ferroptosis assays, subcutaneous xenograft models Cell reports Medium 42207639

Source papers

Stage 0 corpus · 34 papers · ranked by NIH iCite citations
Year Title Journal Citations PMID
2001 Identification and characterization of a family of Rab11-interacting proteins. The Journal of biological chemistry 252 11495908
2009 RCP is a human breast cancer-promoting gene with Ras-activating function. The Journal of clinical investigation 110 19620787
2012 Deregulation of Rab and Rab effector genes in bladder cancer. PloS one 100 22724020
2022 CircRTN4 promotes pancreatic cancer progression through a novel CircRNA-miRNA-lncRNA pathway and stabilizing epithelial-mesenchymal transition protein. Molecular cancer 81 34983537
2013 Rab11-family interacting proteins define spatially and temporally distinct regions within the dynamic Rab11a-dependent recycling system. Molecular biology of the cell 64 23283983
2017 Downregulation of ARID1A, a component of the SWI/SNF chromatin remodeling complex, in breast cancer. Journal of Cancer 34 28123592
2003 Dynamics of the apical plasma membrane recycling system during cell division. Traffic (Copenhagen, Denmark) 33 12956871
2009 Genomic amplicons target vesicle recycling in breast cancer. The Journal of clinical investigation 31 19620778
2006 The Rab11-FIP1/RCP gene codes for multiple protein transcripts related to the plasma membrane recycling system. Biochimica et biophysica acta 30 16920206
2023 Augmenting Intracellular Cargo Delivery of Extracellular Vesicles in Hypoxic Tissues through Inhibiting Hypoxia-Induced Endocytic Recycling. ACS nano 27 36730125
2019 The integral function of the endocytic recycling compartment is regulated by RFFL-mediated ubiquitylation of Rab11 effectors. Journal of cell science 25 30659120
2013 The rab11 effector protein FIP1 regulates adiponectin trafficking and secretion. PloS one 25 24040321
2021 Rab11-FIP1 mediates epithelial-mesenchymal transition and invasion in esophageal cancer. EMBO reports 23 33403789
2020 Components of the endocytic and recycling trafficking pathways interfere with the integrity of the Legionella-containing vacuole. Cellular microbiology 20 32096265
2016 Rab11-FIP1A regulates early trafficking into the recycling endosomes. Experimental cell research 16 26790954
2016 Micro ribonucleic acid-93 promotes oncogenesis of cervical cancer by targeting RAB11 family interacting protein 1. The journal of obstetrics and gynaecology research 16 27279231
2014 Distinct patterns of phosphatidylserine localization within the Rab11a-containing recycling system. Cellular logistics 13 25210648
2017 Rab11-FIP1 phosphorylation by MARK2 regulates polarity in MDCK cells. Cellular logistics 12 28396819
2021 Rab11-FIP1 and Rab11-FIP5 Regulate pIgR/pIgA Transcytosis through TRIM21-Mediated Polyubiquitination. International journal of molecular sciences 10 34638806
2024 Combining genetic and single-cell expression data reveals cell types and novel candidate genes for orofacial clefting. Scientific reports 8 39489835
2022 Rab11FIP1-deficient mice develop spontaneous inflammation and show increased susceptibility to colon damage. American journal of physiology. Gastrointestinal and liver physiology 8 35819177
2008 CagA of Helicobacter pylori alters the expression and cellular distribution of host proteins involved in cell signaling. FEMS microbiology letters 8 18811656
2024 Homodimeric Granzyme A Opsonizes Mycobacterium tuberculosis and Inhibits Its Intracellular Growth in Human Monocytes via Toll-Like Receptor 4 and CD14. The Journal of infectious diseases 7 37671668
2023 The transcription factor Stat-1 is essential for Schwann cell differentiation, myelination and myelin sheath regeneration. Molecular medicine (Cambridge, Mass.) 6 37365519
2021 Protein profiling reveals potential isomiR-associated cross-talks among RNAs in cholangiocarcinoma. Computational and structural biotechnology journal 4 34745457
2024 The circRNA Landscape in Recurrent Pregnacy Loss (RPL): A Comparison of Four Reproductive Tissues. International journal of molecular sciences 3 39684334
2023 Eukaryotic Clathrin Adapter Protein and Mediator of Cholesterol Homeostasis, PICALM, Affects Trafficking to the Chlamydial Inclusion. Molecular and cellular biology 2 36779337
2021 Rab11-FIP1/RCP Functions as a Major Signalling Hub in the Oncogenic Roles of Mutant p53 in Cancer. Frontiers in oncology 2 35757381
2025 Identification and study of mood-related biomarkers and potential molecular mechanisms in type 2 diabetes mellitus. Journal of molecular histology 1 39915429
2025 Proteomics reveals genetic mechanisms of cold resistance in Hezuo pig liver tissue. Journal of proteomics 1 40064419
2025 A pilot study on DNA methylation changes for non-invasive molecular diagnostics in heart failure. ESC heart failure 1 40898655
2023 Ocular and neural genes jointly regulate the visuospatial working memory in ADHD children. Behavioral and brain functions : BBF 1 37658396
2026 Age-related lncRNA alterations of SDHAP2_miR-17-5p/miR-20b-5p_RAB11FIP1 ceRNA network in donor-derived human limbal epithelial cells. Frontiers in cell and developmental biology 0 41983190
2026 USP52-RAB11FIP5 axis suppresses ferroptosis by repressing transferrin receptor recycling in head and neck squamous cell carcinoma. Cell reports 0 42207639

Missed literature

Know a paper Affinage missed for RAB11FIP1? Flag it for the maintainers and the community.

No submissions yet.