| 2000 |
NGEF was identified as a novel member of the Dbl family of guanine nucleotide exchange factors (GEFs), capable of acting as a GEF for Rho-type GTPases. It demonstrated transforming potential in cell culture and ability to induce tumors in nude mice, consistent with GEF/RhoGTPase signaling activity. |
Sequence analysis (Dbl family domain identification), cell transformation assay, nude mouse tumor induction |
Genomics |
Medium |
10777665
|
| 2012 |
In Xenopus convergence and extension cell movements, NGEF acts downstream of RPTPα/PTPε phosphatases to promote RhoA activation. Morpholino knockdown of NGEF disrupted convergence and extension, and this phenotype was rescued by constitutively active RhoA, placing NGEF as a positive regulator of RhoA in this pathway. |
Morpholino knockdown in Xenopus embryos, epistasis with constitutively active/dominant-negative RhoA rescue, co-knockdown specificity controls |
PloS one |
High |
22545146
|
| 2025 |
NGEF (Ephexin1) promotes axonal growth in neurons co-cultured with lung cancer cells via the Ephrin-A3/EphA2 pathway. Knockdown of Ephrin-A3 in neurons or inhibition of EphA2 with ALW-II-41-27 blocked neurite outgrowth stimulated by NGEF-overexpressing cancer cells. In a mouse xenograft model, NGEF overexpression increased tumor growth and nerve fiber density, effects inhibited by ALW-II-41-27. |
Co-culture experiments (neurons + cancer cells), Ephrin-A3 knockdown, EphA2 inhibitor (ALW-II-41-27), mouse subcutaneous xenograft model with nerve fiber density quantification |
Journal of translational medicine |
Medium |
40022166
|
| 2025 |
Ephexin1/NGEF is essential for polysome formation and promotes translation of mRNAs containing 5'-terminal oligopyrimidine (TOP) or 5'-TOP-like motifs, acting as a key mediator of mTOR-regulated translation. Ephexin1 deficiency enhanced efficacy of mTOR inhibitors in a mouse xenograft lung cancer model, indicating synthetic lethality. |
Polysome profiling, gene expression analysis after NGEF knockdown, mouse xenograft model with mTOR inhibitor combination |
Experimental & molecular medicine |
Medium |
40855114
|
| 2025 |
NGEF promotes invasion and migration of BRAFV600E-mutant thyroid cancer cells through the epithelial-mesenchymal transition (EMT) pathway. NGEF expression is transcriptionally regulated by BRAFV600E via the ERK/AP1 pathway, as confirmed by pathway inhibition experiments and dual-luciferase reporter assays. |
Functional invasion/migration assays, EMT biomarker analysis, pathway inhibition, dual-luciferase reporter assay, BRAFV600E-engineered cellular models |
Biochemistry and biophysics reports |
Medium |
40703409
|
| 2025 |
Variants in NGEF were identified in cases of idiopathic scoliosis (IS), and NGEF is positioned as a downstream molecule in the EPHA4 signaling pathway relevant to spinal patterning, based on analysis of IS cohort data. |
Human genetic variant analysis in IS cohorts; pathway placement as downstream of EPHA4 based on known pathway membership |
eLife |
Low |
40662934
|