| 2015 |
CCNE2 is a direct target of miR-30d-5p; miR-30d-5p binds to CCNE2 and suppresses its expression, thereby inhibiting NSCLC cell proliferation, invasion, and migration. Re-introduction of CCNE2 antagonized the inhibitory effects of miR-30d-5p. |
Luciferase reporter assay, Western blot, functional rescue experiments in NSCLC cells |
Cancer letters |
Medium |
25843294
|
| 2015 |
CCNE2 acts downstream of HMGA1 to regulate motility and invasiveness of basal-like breast cancer cells by promoting nuclear localization and activity of YAP (Hippo pathway mediator). MST1/2 and LATS1/2 kinases are required for HMGA1- and CCNE2-mediated regulation of YAP localization. CDK inhibitors induce translocation of YAP from nucleus to cytoplasm. |
Genetic epistasis (knockdown/overexpression), subcellular fractionation/localization of YAP, CDK inhibitor treatment |
Oncotarget |
Medium |
26265440
|
| 2017 |
CCNE2 overexpression contributes to acquired trastuzumab resistance in HER2+ breast cancer; silencing CCNE2 in trastuzumab-resistant BT474r cells restored sensitivity to trastuzumab. miR-26a and miR-30b regulate CCNE2 expression and trastuzumab response. |
siRNA knockdown of CCNE2, cell viability assays, miRNA overexpression in resistant cell lines |
Scientific reports |
Medium |
28120942
|
| 2016 |
miR-26a directly targets the 3' UTR of CCNE2 (and CCND2) to regulate mouse hepatocyte proliferation and cell cycle progression. Inhibition of miR-26a upregulated CCNE2 expression and increased hepatocyte proliferation. |
Dual-luciferase reporter assay, Western blot, flow cytometry, adenoviral overexpression/inhibition in hepatocyte cell line |
Hepatobiliary & pancreatic diseases international |
Medium |
26818545
|
| 2016 |
Beryllium induces p53-dependent transcriptional downregulation of CCNE2 mRNA (~90% reduction). However, reduction in CCNE2 mRNA did not lead to corresponding reductions in cyclin E2 protein, which had an unusually long half-life (>12 hours), indicating post-transcriptional stabilization of CCNE2 protein independent of p53. |
siRNA knockdown of p53, RT-PCR, Western blot, cycloheximide chase, proteasomal inhibition (MG-132) |
Cell proliferation |
Medium |
27611480
|
| 2020 |
CARM1, an arginine methyltransferase, is recruited to the CCNE2 gene promoter and activates CCNE2 transcription. Asymmetric di-methylation marks H3R17me2a and H3R26me2a are enriched at the CCNE2 core promoter. Restoration of CCNE2 abolished CARM1-knockdown-mediated inhibition of cell proliferation, placing CCNE2 downstream of CARM1 in NSCLC. |
ChIP assay, luciferase reporter assay, shRNA knockdown, overexpression rescue experiments in vitro and in vivo |
Aging |
High |
32487779
|
| 2021 |
E2F1 transcription factor directly targets the CCNE2 promoter and activates its transcription in TGF-β1-induced human cardiac fibroblast differentiation. Luciferase reporter assay and immunoprecipitation confirmed CCNE2 as a direct target gene of E2F1. Silencing CCNE2 decreased cardiac fibroblast differentiation. |
Luciferase reporter assay, immunoprecipitation, siRNA knockdown, Western blot |
Bioengineered |
Medium |
34521301
|
| 2021 |
MFA (methyl ferulic acid) attenuates cardiac fibroblast differentiation and myocardial fibrosis by suppressing the pRB-E2F1/CCNE2 pathway (as well as RhoA/ROCK2 pathway), reducing CCNE2-mediated proliferation and migration of human cardiac fibroblasts. |
Western blot, cell migration/proliferation assays, in vivo myocardial infarction mouse model |
Frontiers in pharmacology |
Low |
34483923
|
| 2019 |
miR-30a directly interacts with the CCNE2 3'UTR; nicotine-induced upregulation of miR-30a downregulates CCNE2 expression and arrests human periodontal ligament cells in G1 phase. Inhibition of miR-30a restored CCNE2 expression downregulated by nicotine. |
Luciferase reporter assay, qRT-PCR, cell cycle analysis (flow cytometry), miR-30a inhibitor rescue |
Biochemistry and cell biology |
Medium |
31689122
|
| 2019 |
CircCCNB1 acts as a sponge for miR-449a to maintain CCNE2 expression; reduced CircCCNB1 inhibits CCNE2 expression via miR-449a, repressing cellular proliferation and triggering cellular senescence (increased SA-β-gal, p21, p53). |
Whole-transcriptome sequencing, functional knockdown of CircCCNB1, miR-449a activity assays, SA-β-gal assay, Western blot |
Aging |
Medium |
31767812
|
| 2020 |
circ-CSPP1 acts as a sponge for miR-577, positively regulating CCNE2 (a target of miR-577) in hepatocellular carcinoma. Knockdown of circ-CSPP1 downregulated CCNE2, p-Rb, E2F1, and c-myc. miR-577 inhibitor rescued suppressive effects of circ-CSPP1 knockdown, but was reversed by CCNE2 silencing. |
Dual luciferase assay, RNA immunoprecipitation, RNA FISH, knockdown/overexpression, xenograft model, Western blot |
Cancer cell international |
Medium |
32514247
|
| 2024 |
ELF3 (ETS transcription factor) directly activates the CCNE2 promoter; ELF3 overexpression enhances CCNE2 promoter activity. ELF3 silencing reduces CCNE2 expression, induces cell cycle arrest, and suppresses malignant transformation of HPV16 E6/E7-immortalized keratinocytes. |
Dual luciferase reporter assay (promoter activity), bioinformatics binding site analysis, siRNA knockdown, CCK-8 assay, cell cycle analysis, Western blot |
Journal of microbiology and biotechnology |
Medium |
39572025
|
| 2026 |
Knockdown of CCNE2 in rheumatoid arthritis synovial fibroblasts (RASFs) induced cellular senescence (elevated p16, p21, p53, SA-β-gal activity, H3K9me3) and apoptosis, and reduced cell viability. In vivo AAV-mediated intra-articular shCCNE2 reduced arthritis index and joint inflammation in CIA mice, also enhancing SASP factor secretion (MMP-3, IL-8). |
shRNA knockdown, flow cytometry (apoptosis), SA-β-gal assay, Western blot, AAV intra-articular injection in CIA mouse model |
Modern rheumatology |
Medium |
40820921
|
| 2024 |
CircHAS2 promotes CCNE2 activation via two axes: (1) sponging miR-1244 to relieve repression of CCNE2, and (2) binding USP10 to facilitate p53 nuclear export and degradation, thereby de-repressing CCNE2 expression. This bidirectional regulation promotes colorectal cancer cell proliferation. |
RNA sequencing, molecular biology (luciferase, RIP), patient-derived organoids, xenograft models |
Molecular cancer |
Medium |
38515149
|
| 2020 |
Tigecycline suppresses CCNE2 expression in pancreatic ductal adenocarcinoma cells; CCNE2 overexpression significantly rescued tigecycline-inhibited cell proliferation and migration/invasion, placing CCNE2 as a downstream effector of tigecycline's anti-tumor action. |
Western blot, CCNE2 overexpression rescue, MTT/BrdU/soft agar assay, wound healing, transwell assay, xenograft model |
Journal of cellular and molecular medicine |
Low |
32141702
|